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This is achieved by inhibiting monoamine oxidation and blocking the reuptake of 5-HT and NE, thereby increasing extracellular levels of 5-HT, DA, and NE throughout the brain

To investigate whether albumin could be engineered to evade L585 cleavage and the resulting reduction in FcRn binding, we constructed a panel of 18 full-length 585-engineered human albumin variants, in which the C-terminal leucine residue was substituted with each of the other naturally occurring amino acids, except for cysteine (Fig

What remains speculative or early-stage: Specific peptide stacking protocols for neuropathy have not been tested in controlled clinical trials

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